
Natural neuroprotective agent on glutamate-mediated HT22 клетка death, controlling the amount of ubiquinone, up-regulating p21 expression in a p53-dependent manner, decreasing Akt phosphorylation and resulting in increased Fas лиганд production and caspase-
Natural neuroprotective agent on glutamate-mediated HT22 cell death, controlling the amount of ubiquinone, up-regulating p21 expression in a p53-dependent manner, decreasing Akt phosphorylation and resulting in increased Fas ligand production and caspase-
Только для научно-исследовательского применения. Не предназначено для использования у пациентов или в качестве лекарственного средства.